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Oud 9 July 2008, 14:22   #1
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fruitsuikers  

Kunnen suikers in fruit eigenlijk veel kwaad in je diëet ?

Wanneer eet je ze het beste ?

Kan iemand me daar eens wat meer uitleg over geven , want wanneer ik bvb lees welke voedingswaarde bvb een mango heet dan lees ik .

per 100 gram verteerbare KH 14,1 gram suikers 12,2 ?

Sommige zeggen dat fruitsuikers weinig kwaad kunnen , andere zeggen weer van wel .

En ik ben een grote fruiteter .
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Oud 9 July 2008, 14:25   #2
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Re: fruitsuikers  

Typ eens 'fructose' in bij de zoek-functie.
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Oud 9 July 2008, 14:26   #3
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Re: fruitsuikers  

http://dikkepret.web-log.nl/dikkepre...uiker_sle.html

Imo geen reden om fruit te laten. Ik eet elke dag 5 stukken fruit, gemixt tussen mijn kwark

Ik ken trouwens veel meer mensen die dik geworden zijn van koekjes en gebak dan van fruit
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Oud 9 July 2008, 16:52   #4
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Re: fruitsuikers  

Citaat:
Origineel gepost door maiskorrel Bekijk Post
http://dikkepret.web-log.nl/dikkepre...uiker_sle.html

ik ken trouwens veel meer mensen die dik geworden zijn van koekjes en gebak dan van fruit
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Oud 9 July 2008, 16:55   #5
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Re: fruitsuikers  

Vreemd onderzoek waar op die site over gepraat wordt. Muizen die frisdrank dronken tov muizen die vruchtensap dronken. En ik maar denken dat diertjes als muizen niet tegen de bubbeltjes kunnen...
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Oud 9 July 2008, 18:17   #6
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Re: fruitsuikers  

Citaat:
Origineel gepost door Woopy Bekijk Post
Vreemd onderzoek waar op die site over gepraat wordt. Muizen die frisdrank dronken tov muizen die vruchtensap dronken. En ik maar denken dat diertjes als muizen niet tegen de bubbeltjes kunnen...
dan laten ze hele lieve kleine boertjes denk ik ?
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Oud 9 July 2008, 18:44   #7
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Re: fruitsuikers  

Kunnen ze niet voor zover ik weet, en dat is ook de reden dat ze er niet tegen kunnen.

edit:
ow ik snap het. de frisdrankmuizen zijn gewoon gestorven. geen wonder dat ze lichter waren dan de fructosemuizen!
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Oud 9 July 2008, 19:44   #8
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Re: fruitsuikers  

http://www.parrillo.com/publications/45.pdf blz 8, fat-forming fructose.


http://www.parrillo.com/sngdetails.asp?sng=tsb&id=119


fruit
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Laatst aangepast door 3XL : 9 July 2008 om 19:52.
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Oud 9 July 2008, 19:44   #9
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Re: fruitsuikers  

Fructose- The Evil SugarBy: Brian Gurneak

No way you say, how could something in fruit make me fat? Doesn't fruit have a lot of vitamins/minerals, fiber and water to make it a healthy snack? I thought you were so adamant about whole foods, Brian?

Okay, hold the boat here a second. Let me explain to you in detail what I mean.

Way back in the mid 1990's while I was contemplating doing my first bodybuilidng contest, I was under the assumption that as long as I ate my fruit and veggies often I would develop into a lean machine. Fact is I was always thin, and thought I could NEVER gain bodyfat. So here I took out all the "junk" foods, and you guys know what they are and implemented more fruit. Soon after I began this process, I started to notice my body didn't change much, in fact my bodyfat went up a little. I was doing my aerobic and lifting weights constantly, but still little change.

Then one day, someone told me about a company called Parrillo Performance and that I should read this publication the person handed to me. Throughout the publication it discussed many articles on training and supplements and one on what do you know FRUIT, and how it's not good for someone who wants to lose bodyfat lbs. Shocked by the article, and how I deeesperately wanted to LOSE bodyfat lbs, (it was approaching summer), I had to give it a try.

Well, roughly 4-5 weeks later I noticed a difference in the way I looked. Tighter in the belly area, was the obvious difference just because I lowered greatly the amount of fruit in my diet. Now let me tell you the reason why, and give you some pointers. Fruit is made up of a simple sugar known as fructose. Bottom line is fructose doesn't convert to be used as a storage sugar efficiently like starchy carbs (rice oats, peas, corn) meaning it is easily converted to bodyfat. Who the heck wants that? I am not going into the biochemistry of enzymes and the like, it will quite frankly bore you. I am not saying it isn't healthy and nutritious for your body, but if YOU want to cut bodyfat and lean out reduce the fruit intake in your diet.

Tips and Suggestions on fruit:
1. Eat fruits that contain less fructose. (Pears, Granny smith apples, strawberries, blackberries, blueberries, kiwi, Macintosh apples)
2. Avoid high-fructose based fruits for bodyfat loss (raisins, pineapple, watermelon, oranges)
3. The BEST time you can have ANY type of fruit is after of an hour of INTENSE exercise to replenish lost energy from muscles and liver, and to improve recovery for the next workout. Keep in mind you should have a serving or two of protein mix with that fruit.
4. Eat more of a variety of fibrous veggies, especially dark, green leafy veggies( broccoli, green beans, spinach, zucchini etc.), instead of fruit to get the same vitamin/mineral, fiber, and water benefit.


----------------------------------------------------------------------------


Article of the Week: Why We Don't Like Fruit


You’d have thought John Parrillo was the Grinch that Stole Christmas: he has never made a more unpopular pronouncement than his statement that fruit has no place in the dietary game plan of a bodybuilder, or for that matter, anyone else interested in losing body fat. He gets more grief over this statement than anything else he’s ever said, with the possible exception of “a man can eat 10,000 calories a day and not get fat.” So Parrillo hates fruit: that’s like kicking a puppy or telling a schoolroom full of 5-year olds that Santa Claus is just a jive myth perpetrated by The Man in order to create a capitalistic windfall. People new to Parrillo precepts and philosophies are dumbfounded. “No fruit! Every fitness expert on the face of the earth makes fruit one of the cornerstones of a sound nutritional game plan.” John remains unfazed in the face of the fruit backlash: After all, his dissing of fruit and banning it altogether from the Parrillo Nutritional Plan is not based on preferential hatred or personal dislike, it’s based on science and biology and insulin. “Don’t get me wrong,” John said a few years back, “Fruit is generally a healthy, nutritious food, high in fiber, low in fat and a good source of vitamins and minerals. However, for bodybuilders intent on achieving as low a body fat percentile as possible and for anyone trying to maximize fat loss, fruit is a disaster! I discovered this unfortunate fact conducting carefully controlled nutritional experiments with bodybuilders preparing for competition. I found that in case after case, when fruit was added to their strict diet regimen, they added body fat. Conversely when bodybuilders preparing for competition removed fruit from their strict eating regimen, they lost body fat. I knew something about fruit consumption was counterproductive to losing body fat.”

What Parrillo determined in less time than it takes to say, “Got Fruit!” was that 80 to 90% of all the calories contained in fruit are derived from simple sugars: fructose and glucose. Fructose is rapidly converted into body fat by the liver. Whereas most other carbohydrate sources are preferentially stored as glycogen, fructose is preferentially converted to body fat and stored in adipose tissue. This is bad news for the fruit fanatic intent on getting ripped. The molecular structure of fructose allows it to skip a key regulatory point in carbohydrate metabolism, a step in the glycoltic pathway catalyzed by the phosphofructokinase-1 or PKF-1 for short. That’s a hell of a mouthful. PKF-1 is the enzyme that regulates if dietary energy supplied by carbohydrates is used for energy or is stored, either as glycogen or as body fat. PKF-1 shuttles carbs into glycogen storage until the tanks are full. At that point PFK-1 will start shuttling carbohydrate into fat storage. The amount of carbs that can be stored as glycogen is quite limited, generally believed to be about 250 to 400 grams depending upon body weight. That amounts to 1000 to 1600 calories, not much. Fructose skips all this and has the horrific ability to be converted into body fat by the liver. Even if you had room in your glycogen storage tank, fructose would bypass that and go directly to body fat. What a revolting development.

Fructose is absorbed by the small intestine and directly transported to the liver via the portal vein. The first enzyme to act is fructokinease. By the way: guess what the number one sweetner used in commercially available “sports nutrition” bars is? You guessed it: fructose! We did a few surveys a few years back and discovered that out of 26 bars we tested, 25 used fructose as their sweetener and used it in huge amounts. Fructose was usually the first or second ingredient on the label. Corn syrup and fruit juice are cheap and very sweet. The bar makers that load up on fructose point to the fact that it has a low glycemic rating; this is a BS slight-of-hand: the reason fructose has a low GI rating is because it doesn’t even have a chance to spike insulin – it’s in too much of a hurry to bypass the carb highway on its way directly into body fat storage! Remember how fructose bypasses the regular carbohydrate pathways? Well that’s precisely why it has a low GI rating. The body treats it like a fat, and like saturated fat fructose just heads right into body fat stores assuming the body is in a calorie-plus metabolic status. This is why we at Parrillo Performance have a bad opinion of fruit: it is loaded with fructose. Our repeated in-the-trenches experience with competitive bodybuilders has shown us repeatedly that fruit adds body fat to those trying to lower their body fat percentiles. Nothing personal against this dietary mainstay, but if you are interested in losing as much body fat as possible – toss the fruit!
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Oud 9 July 2008, 19:49   #10
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Re: fruitsuikers  

FRUCTOSEby Lyle McDonald


As with so many other aspects of nutrition, especially bodybuilding and sports nutrition, beliefs about the sugar fructose (more commonly known as fruit sugar) vary widely. On the one hand, because of its low glycemic index (GI) and general lack of insulin response, many people consider fructose an ideal sugar, that should be used to replace other sugars especially for diabetics (1). There is also some indication that fructose may blunt appetite and affect food choice (2,3). Finally, because of the low insulin response, it's been suggested that fructose before or during exercise might allow increased fat utilization during exercise, while still maintaining blood glucose levels (4).

At the other end of the spectrum, because of differences in its metabolism compared to other sugars, and the known effect of fructose on blood triglyceride (fat) levels, many nutritional authorities (most notably John Parillo) consider fructose and fruit a sort of nutritional satan that will only make you fat and that should be eliminated from the diets of bodybuilders

What Is Fructose, How Much Do We Get and
Where Is It Found In the Diet?

Fructose is one of three monosaccharides (single sugar molecules, the other two are glucose and galactose) that occurs naturally in foods. Sucrose (table sugar, a disaccharide) is also 1/2 fructose. However, free fructose only occurs naturally in a few foods, notably some fruits (hence its common name of 'fruit sugar') and honey with the majority of our dietary fructose coming from the ingestion of sucrose. Fructose is also found in small amounts in a few vegetables.

In recent years, the amount of fructose being consumed has increased significantly. One large scale analysis puts average daily amounts in the range of 90-100 grams (8) which is a significant increase over the last 2 decades. This increase has been caused not only by an overall increase in the consumption of sugars, but also because of increased use of high fructose corn syrup (HFS). HFS contains can contain 42% or 55% free fructose, with the remainder being glucose. So, on top of an overall increase in fructose consumption, the use of HFS has caused a significant increase in the consumption of free fructose (8,9).

Additionally, many health-food stores also sell crystalline fructose powder as a sweetener and both crystalline fructose and HFS are used in many commercially produced food products (10).

Fructose Digestion and Metabolism

During digestion in the stomach and small intestines, all dietary carbohydrates are eventually broken down to the monosaccharides glucose, fructose and galactose (found in milk in lactose). These are absorbed via specific transporters in the small intestine, bringing them into the portal vein, next stop the liver.

While the small intestine seems to have an essentially unlimited (estimated around 5000 grams per day) capacity to absorb glucose and galactose, the absorption of fructose is a very different story. The consumption of as little as 35-50 grams of free fructose at once causes gastric upset, gas, bloating and diarrhea in a majority (60% or more) of people (11). However, the addition of glucose to the free fructose prevents the problem. And the ingestion of large amounts of sucrose (again, 1/2 fructose, 1/2 glucose) causes no such problem. It thus appears that our guts have evolved to absorb fructose only in the presence of other sugars and that large amounts of free fructose in the diet are non-physiological for humans. Since HFS also contains a significant amount of glucose, the malabsorption issue may not be a huge one for most processed foods. However, anyone considering using crystalline fructose as a sweetener should consider the potential problems with large amounts of free fructose. I should also mention that some individuals suffer from a hereditary fructose malabsorption syndrome (12), but this is typically identified at a very young age. Fructose free diets can be developed for these individuals.

After digestion, fructose goes to the liver like all sugars, which is where the bulk of its metabolism occurs. This is also where fructose metabolism differs significantly from glucose metabolism, and is the source of much of the debate over the relative 'goodness' of fructose.

Liver Metabolism of Sugars

Although they share many intermediate steps in their metabolism, dietary glucose and fructose follow two distinct pathways in the liver. In fact, it has been known for quite some time that most dietary glucose goes straight through the liver with only minimal metabolism (13) while the majority of fructose is metabolized in the liver (14).

This is part of the reason for the low glycemic index (GI) of fructose. Since it is metabolized almost exclusively in the liver, it has only a very small effect on blood glucose levels. Additionally, and also unlike glucose, the uptake of fructose into liver cells doesn't require insulin. Hence there is no need for the body to secrete insulin in response to dietary fructose ingestion. This is the basis for the claims that fructose is a superior carbohydrate source compared to glucose or sucrose (which recall is half glucose), especially for diabetics (1) who have poor control over blood glucose and insulin levels.

At the same time, because of its extensive metabolism, and the pathways that it follows, in the liver, fructose can have negative effects on the overall metabolism of the body. So first let's compare and contrast the metabolism of glucose and fructose in the liver, and then look at some of the potential negatives of fructose (especially excess fructose) intake.

But Just How Much Are We Talking About?

A cursory examination of the research into fructose feedings in terms of the production of metabolites such as lactate, uric acid and especially VLDL triglycerides has shown distinctly varying results. For example, while some research has clearly shown an increase in VLDL triglyceride levels with fructose feedings (18-23), other research has not (24-28). Why the discrepancy?

As with so many aspects of nutrition, it really comes down to two things: how much fructose they gave, and what potion they gave it to. Let's get the potion dynamic out of the way first. Studies have examined the effects of fructose in essentially 4 different groups: individuals with normal insulin and triglyceride levels, individuals with normal insulin but high triglyceride levels, individuals with high insulin and normal triglyceride levels, and people with high insulin and triglyceride levels. Different groups show a different susceptibility to the negative effects of fructose (and that still has to take amount into account).

Overall, the last group, those with high insulin and high triglyceride levels tends to be the most sensitive to the negative effects of fructose in terms of increasing VLDL triglyceride levels (3). The other three groups show a distinctly less pronounced effect. This makes some sense as such individuals would normally have skewed physiology to begin with. I should point out that hyperinsulinemic/hypertriglyceridemic folks aren't very indicative of the average lean athlete consuming a healthy bodybuilding/sports oriented diet.

But that brings us back to dose. Even in otherwise healthy individuals, fructose has been found to increase VLDL triglyceride levels so the potential for fat synthesis from fructose is apparently there. Again, looking at the studies as a whole, both negative and positive results are typically found. It's when you start looking at the amounts given, that a pattern starts to develop. First, a select group of studies has used absurd and non-physiological amounts of fructose (200-500 grams per day, more than any human is probably capable of consuming under all but the most forced conditions) and invariably found increased triglyceride levels. Keep in mind that the average American diet only contains about 30-40 grams of fructose per day so we can pretty safely ignore those studies.

Looking at the other studies which gave more reasonable amounts of fructose, as a recent review has done (29), we see a fairly standard pattern: at reasonable amounts of fructose (30-60 grams per day depending on the study), there is no negative effect on VLDL or triglyceride levels. At amounts higher than that (in the range of 80-90+ grams per day), there tend to be an increase in VLDL and triglyceride levels suggesting fat synthesis. This would tend to suggest a distinct cutoff point somewhere between those two values as an approximate maximum of fructose that can be consumed without causing significant triglyceride synthesis.

In contrast, one study comparing 75 grams of fructose to 75 grams of glucose, found that, over 4 hours of study, while there was a small amount of de novo lipogenesis from the fructose, the net effect was that the body burned more fat than it produced (30). The fructose group also showed a higher thermic effect (meaning more calories were wasted as heat), most likely because of the high amount of metabolic processing that went on. However, and perhaps more importantly, despite very little fat synthesis in the fructose group, there was less fat burning in that same group. This occurred with an increased burning of carbohydrate in the fructose group.

So it may be that, while fructose at moderate (<50 g/day or so) amounts doesn't increase fat synthesis per se significantly, it may slow fat loss by decreasing fat burning in the liver. That is, to a degree, the end result may be the same: whether the fructose is causing more fat synthesis, or less fat burning, the net effect on fat loss (which is determined by fat burning - fat intake) may be similar.

As I mentioned above, the metabolic fate of fructose appears to depend on the metabolic state of the liver and the dieter. In the fasted state (as occurs while dieting), fructose will be used for energy and fat synthesis will be negligible, if it occurs at all. Fat burning may be decreased however. In the fed state (as occurs when not dieting), excess fructose can be converted to VLDL TG, increasing heart disease risk and bodyfat.

Fructose Feeding and Exercise Performance

Finishing up, I want to touch on the idea of fructose feeding during exercise. To my knowledge, no studies have examined the consumption of different types of carbohydrates prior to weight training, and all of the research done to date has been in endurance athletes. From a theoretical standpoint (including the low GI/insulin response), there are some good reasons to think that fructose feedings might be superior to glucose during endurance exercise. However, the research to date has not supported the theory. Fructose feeding before or during has been found to be either no better, or in fact worse in terms of performance of endurance exercise (3). As well, there is the issue of gastric upset with high amounts of fructose that limits how much can be given in the first place.

As a final comment, there is the issue of post-workout carbohydrate and recovery. At this point, it should be no surprise to readers that the post-workout consumption of carbs and protein improves recovery, performance and protein synthesis (32,33). Studies examining different types of carbohydrate intake post workout have invariably found that glucose and glucose polymers refill muscle glycogen ideally, while fructose preferentially refills liver glycogen. In that refilling liver glycogen can be important from the standpoint of overall recovery and growth (the details are outside of the scope of this article), consuming a small amount of fructose (10% of the total carbohydrate content or roughly 10-20 grams) in the post-workout shake may be beneficial. But the majority of carbohydrates consumed should come from glucose and glucose polymers.

Summing Up and Practical Recommendations

Ok, an article like this wouldn't be worth much without some actual recommendations and real world application. First and foremost, it should be clear that in large enough amounts, fructose can certainly be detrimental both to health, by raising VLDL cholesterol and triglycerides and possibly to bodyfat levels for the same reason. However, at moderate intakes of fructose, in the range of 50-60 grams per day, fructose appears to pose little problem and certainly is not going to make or break a diet.

For an athlete to avoid all sources of fructose, especially fruit, seems a bit silly and extreme (see below regarding pre-contest bodybuilding prep for a possible exception). However, there is probably a good reason to avoid high fructose corn syrup as much as is reasonably possible. Readers should realize that many sports food companies are using fructose and HFS in their products, so it's possible that athletes are being exposed to larger than normal amounts of fructose in their diets. Athletes and bodybuilders are encouraged to become avid label readers to see if HFS is listed as a primary ingredient.

Individuals who are hyperinsulinemic or have high triglycerides to begin with may question whether using fructose in large amounts is beneficial and should consult with their doctor before making major nutritional changes. The majority of athletes, bodybuilders and otherwise healthy individuals are unlikely to have problems with either hyperinsulinemia or hypertriglyceridemia although it is a possibility.

So, back to the ~50 g/day value. Noting that the average American diet may contain at least double that already, we might assume that athletes shouldn't be adding more fructose or fruit to their diet. But, we really have to ask whether or not that applies to bodybuilders and athletes, who typically avoid the commercial foods which most commonly containing fructose (meaning those containing high fructose corn syrup).

Most bodybuilders and other athletes already avoid the majority of such foods and I would expect that their daily fructose intake is somewhat below the American average. Once again, note that many commercial products aimed at athletes, such as food bars and even some meal replacement powders, are increasing their use of fructose and HFS as a sweetener so it is possible that athletes are getting more fructose or HFS than they're aware of. How much is up to debate and speculation.

So let's address the important question: What about fruit? Can it be part of a healthy bodybuilding/athletic diet, or should it be avoided as Parillo claims? To answer this we really need to look at the amount of fructose found in typical fruits.

On average, fruits such as cherries, pears, bananas, grapes and apples contain anywhere from 5 to 7 grams of fructose in an average sized piece of fruit. Fruits such as strawberries, blueberries, oranges and grapefruit contain 2-3 grams of fructose per 100 gram serving. Honey is an exception, containing 40 grams of fructose per 100 gram serving, but its extreme sweetness would make eating a lot of it difficult. The point being that fruit is actually not a very large source of fructose in the first place. To get 50 grams of fructose per day from fruit alone would require an intake of approximately 10 pieces per day, far more than all but the most extreme intake would provide.


So, summing up, like most aspects of bodybuilding and athletic nutrition, there are few absolutes. While there is no doubt that large amounts of fructose are both non-physiological and potentially harmful, it certainly appears that low to moderate amounts of fructose, and yes fruit, can be included in a bodybuilding or athletic diet. From the standpoint of liver glycogen and maintaining an anabolic state, small amounts of fructose probably should be included in the diet. Considering the other nutrients (fiber, vitamins and minerals) present in fruit, it seems silly to exclude them from the diet based on the rather small amount of fructose present


References cited:

1. Gerrits, PE and Tsalikian, E. Diabetes and fructose metabolism. Am J Clin Nutr (1993) 58 (suppl): 796s-799s.

2. Moyer, AE and Rodin, J. Fructose and behavior: does fructose influence food intake and macronutrient selection. Am J Clin Nutr (1993) 58 (suppl): 810s-814s.

3. Henry, RR and o, PA. Current issues in fructose metabolism. Ann Rev Nutr (1991) 11: 21-39.

4. Craig, BW. The influence of fructose feeding on physical performance. Am J Clin Nutr (1993) 58 (suppl): 815s-819s.

4a. Web article at: http://www.parrillo.com/sng/tsb_det...946114869&id=45

5. O'Dell, BL. Fructose and mineral metabolism. Am J Clin Nutr (1993) 58: 771s-778s.

6. Dills, Jr. WL. Protein fructosylation: fructose and the Maillard reaction. Am J Clin Nutr (1993) 58 (suppl): 779s-787s.

7. Glinsmann WH and Bowman, BA. The public health significant of dietary fructose. Am J Clin Nutr (1993) 58 (suppl): 820s-823s.

8. http://www.ers.usda.gov:80/publicat...B965/sb965h.pdf Table 2.

9. Vuilleumier, S. Worldwide production of high-fructose syrup and crystalline fructose. Am J Clin Nutr (1993) 58 (suppl): 733s-736s.

10. Hanover, ML and White, JS. Manufacturing, composition, and applications of fructose. Am J Clin Nutr (1993) 58 (suppl): 724s-732s.

11. Riby, JE et. al. Fructose absorption. Am J Clin Nutr (1993) 58 (suppl): 748s-753s.

12. Ali, M, et. al. Heriditary Fructose Intolerance. J Med Genet (1998) 35:353-365.

13. McGarry JD et. al. From dietary glucose to liver glycogen: the full circle around. Ann Rev Nutr (1987) 7:51-73.

14. Mayes, PA. Intermediate metabolism of fructose. Am J Clin Nutr (1993) 58 (suppl): 754s-765s.

15. De Jong M et. al. T4 uptake into the perfused rat liver and liver T4 uptake in humans are inhibited by fructose.Am J Physiol (1994) 266(5 Pt 1):E768-E775

16. Hellerstein MK. Synthesis of fat in response to alteration in diet: insights from new stable isotope methodologies. Lipids (1996) 31 (suppl): S117-S125.

17. Acheson KJ et. al. Glycogen storage capacity and de novo lipogenesis during massive carbohydrate overfeeding in man. Am J Clin Nutr (1988) 48: 240-247.

18. Jeppesen, J et. al. Postprandial triglyceride and retinyl ester responses to oral fat: effects of fructose. Am J Clin Nutr (1995) 61: 787-791.

19. Abraha, A et. al. Acute effect of fructose on postprandial lipaemia in diabetic and non-diabetic subjects. Br J Nutr (1998) 80: 169-175.

20. Reiser, S et. al. Blood lipids, lipoproteins, apoproteins, and uric acid in men fed diets containing fructose or high-amylose cornstarch. Am J Clin Nutr (1989) 49: 832-839.

21. Bantle, JP et. al. Metabolic effects of dietary fructose in diabetic subjects. Diabetes Care (1992) 15: 1468-1476.

22. Bantle, JP et. al. Effect of dietary fructose on plasma lipids in healthy subjects. Am J Clin Nutr (2000) 72: 1128-1134.

23. Swanson, JE et. al. Metabolic effects of dietary fructose in healthy subjects. Am J Clin Nutr (1992) 55: 851-856.

24. McAteer EJ et. al. The effects of one month high fructose intake on plasma glucose and lipid levels in non-insulin-dependent diabetes. Diabetic Medicine (1987) 4: 62-64.

25. Grigorscro, C et. al. Lack of detectable deleterious effects on metabolic control of daily fructose ingestion for 2 Mo in NIDDM patients. Diabetes Care (1988) 11: 546-550.

26. Eunsook, TK et. al. Effects of fructose feeding on blood parameters and blood pressure in impaired glucose-tolerant subjects. J Am Diet Assoc (1988) 88: 932-938.

27. Koivisto VA and Yki-Jarvinen H. Fructose and insulin sensitivity in patients with type 2 diabetics. J Internal Med (1993) 233: 145-153.

28. Malerbi, DA et. al. Metabolic effects of dietary sucrose and fructose in Type II diabetic subjects. Diabetes Care (1996) 19: 1249-1256.

29. Hollenbeck, CB. Dietary fructose effects on lipoprotein metabolism and risk for coronary heart disease. Am J Clin Nutr (1993) 58 (suppl): 800s-809s.

30. Tappy, L, et. al. Comparison of the thermogenic effect of fructose and glucose in normal humans. Am J Physiol (1986) 250: E718-E724.

31. elbach, TJ, et. al. Post-exercise substrate utilization after a high glucose vs. high fructose meal during negative energy balance in the obese. Obes Res (2000) 8: 496-505.

32. Zawadzki et al. Carbohydrate-protein complex increases the rate of muscle glycogen storage after exercise. J Appl Physiol (1992) 72: 1854-1859.

33. Rasmussen BB et. al. An oral essential amino acid-carbohydrate supplement enhances muscle protein anabolism after resistance exercise. J Appl Physiol. (2000) 88:386-92.
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Oud 10 July 2008, 00:16   #11
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Re: fruitsuikers  

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Article of the Week: Why We Don't Like Fruit


You’d have thought John Parrillo was the Grinch that Stole Christmas: he has never made a more unpopular pronouncement than his statement that fruit has no place in the dietary game plan of a bodybuilder, or for that matter, anyone else interested in losing body fat. He gets more grief over this statement than anything else he’s ever said, with the possible exception of “a man can eat 10,000 calories a day and not get fat.” So Parrillo hates fruit: that’s like kicking a puppy or telling a schoolroom full of 5-year olds that Santa Claus is just a jive myth perpetrated by The Man in order to create a capitalistic windfall. People new to Parrillo precepts and philosophies are dumbfounded. “No fruit! Every fitness expert on the face of the earth makes fruit one of the cornerstones of a sound nutritional game plan.” John remains unfazed in the face of the fruit backlash: After all, his dissing of fruit and banning it altogether from the Parrillo Nutritional Plan is not based on preferential hatred or personal dislike, it’s based on science and biology and insulin. “Don’t get me wrong,” John said a few years back, “Fruit is generally a healthy, nutritious food, high in fiber, low in fat and a good source of vitamins and minerals. However, for bodybuilders intent on achieving as low a body fat percentile as possible and for anyone trying to maximize fat loss, fruit is a disaster! I discovered this unfortunate fact conducting carefully controlled nutritional experiments with bodybuilders preparing for competition. I found that in case after case, when fruit was added to their strict diet regimen, they added body fat. Conversely when bodybuilders preparing for competition removed fruit from their strict eating regimen, they lost body fat. I knew something about fruit consumption was counterproductive to losing body fat.”

What Parrillo determined in less time than it takes to say, “Got Fruit!” was that 80 to 90% of all the calories contained in fruit are derived from simple sugars: fructose and glucose. Fructose is rapidly converted into body fat by the liver. Whereas most other carbohydrate sources are preferentially stored as glycogen, fructose is preferentially converted to body fat and stored in adipose tissue. This is bad news for the fruit fanatic intent on getting ripped. The molecular structure of fructose allows it to skip a key regulatory point in carbohydrate metabolism, a step in the glycoltic pathway catalyzed by the phosphofructokinase-1 or PKF-1 for short. That’s a hell of a mouthful. PKF-1 is the enzyme that regulates if dietary energy supplied by carbohydrates is used for energy or is stored, either as glycogen or as body fat. PKF-1 shuttles carbs into glycogen storage until the tanks are full. At that point PFK-1 will start shuttling carbohydrate into fat storage. The amount of carbs that can be stored as glycogen is quite limited, generally believed to be about 250 to 400 grams depending upon body weight. That amounts to 1000 to 1600 calories, not much. Fructose skips all this and has the horrific ability to be converted into body fat by the liver. Even if you had room in your glycogen storage tank, fructose would bypass that and go directly to body fat. What a revolting development.

Fructose is absorbed by the small intestine and directly transported to the liver via the portal vein. The first enzyme to act is fructokinease. By the way: guess what the number one sweetner used in commercially available “sports nutrition” bars is? You guessed it: fructose! We did a few surveys a few years back and discovered that out of 26 bars we tested, 25 used fructose as their sweetener and used it in huge amounts. Fructose was usually the first or second ingredient on the label. Corn syrup and fruit juice are cheap and very sweet. The bar makers that load up on fructose point to the fact that it has a low glycemic rating; this is a BS slight-of-hand: the reason fructose has a low GI rating is because it doesn’t even have a chance to spike insulin – it’s in too much of a hurry to bypass the carb highway on its way directly into body fat storage! Remember how fructose bypasses the regular carbohydrate pathways? Well that’s precisely why it has a low GI rating. The body treats it like a fat, and like saturated fat fructose just heads right into body fat stores assuming the body is in a calorie-plus metabolic status. This is why we at Parrillo Performance have a bad opinion of fruit: it is loaded with fructose. Our repeated in-the-trenches experience with competitive bodybuilders has shown us repeatedly that fruit adds body fat to those trying to lower their body fat percentiles. Nothing personal against this dietary mainstay, but if you are interested in losing as much body fat as possible – toss the fruit!
interessant, dit is ook echt waar of zijn er onderzoekers die tegenover gestelde dingen aantonen?

ok lees net in die laatste dat het pas vervelend zou kunnen worden boven de 50 gram fructose per dag en dat een banaan maar 6 gram bevat, als ik een banaan op zoek staat er 30gram koolhydraten, wat zijn die andere 24gram koolhydraten in een banaan dan, als maar 6 gram fructose is?

en wat is corn syrup in het nederlands?
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Oud 10 July 2008, 07:53   #12
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Re: fruitsuikers  

Nutrition Facts and Analysis for Bananas, raw

Per 100gr.

Total Carbohydrate 22.8 g 8%
Dietary Fiber 2.6 g 10%
Starch 5.4 g
Sugars 12.2 g
Sucrose 2390 mg
Glucose 4979 mg
Fructose 4850 mg
Lactose 0.0 mg
Maltose 10.0 mg
Galactose 0.0 mg



http://en.wikipedia.org/wiki/Corn_syrup

Corn syrup is maissiroop. Waar je trouwens op moet letten is niet Corn syrup op zich, maar High Fructose Corn syrup (HFCS)
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Oud 10 July 2008, 07:55   #13
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Re: fruitsuikers  

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en wat is corn syrup in het nederlands?
maïsstroop
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Re: fruitsuikers  

(high fructose)maïs siroop? nog nooit van gehoord , is dat ook echt onder die naam verkrijgbaar hier in NL? zie wel overal dextrose op staan



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Nutrition Facts and Analysis for Bananas, raw

Per 100gr.

Total Carbohydrate 22.8 g 8%
Dietary Fiber 2.6 g 10%
Starch 5.4 g
Sugars 12.2 g
Sucrose 2390 mg
Glucose 4979 mg
Fructose 4850 mg
Lactose 0.0 mg
Maltose 10.0 mg
Galactose 0.0 mg


een banaan heeft dus ook gewone suiker erin zitten, wist ik niet...dacht altijd puur fructose, maar dan moet een banaan de insuline spiegel toch wel redelijk wat verhogen? of mis ik iets
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BP: 5 x 40
DL: 2 x 65
quarter Squat: 3 x 55
+ _______
160
status : squats+macdonalds, on the road to big
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Oud 10 July 2008, 09:38   #15
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Re: fruitsuikers  

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(high fructose)maïs siroop? nog nooit van gehoord , is dat ook echt onder die naam verkrijgbaar hier in NL? zie wel overal dextrose op staan
High Fructose Corn Syrup


Ik eet overigens behoorlijk veel bananen op een dag, en word er totaal niet dik van. Bevat ook ongeveer 120kcal per banaan, ik eet er meestal 5 á 6 op een dag.
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Re: fruitsuikers  

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High Fructose Corn Syrup


Ik eet overigens behoorlijk veel bananen op een dag, en word er totaal niet dik van. Bevat ook ongeveer 120kcal per banaan, ik eet er meestal 5 á 6 op een dag.
Bokito wat je bent..
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Re: fruitsuikers  

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High Fructose Corn Syrup


Ik eet overigens behoorlijk veel bananen op een dag, en word er totaal niet dik van. Bevat ook ongeveer 120kcal per banaan, ik eet er meestal 5 á 6 op een dag.
interessant artikel
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Spoiler: Toon

1m83-->65 kg BF 22%
BP: 5 x 40
DL: 2 x 65
quarter Squat: 3 x 55
+ _______
160
status : squats+macdonalds, on the road to big
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Oud 10 July 2008, 14:14   #18
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